> It is a complex answer, and heavily depends on decay product chemistry.
The source you provided does not agree with that assessment. It is purely radiation damage. The Pu-238 (half life 87.7 years) and Pu-239 (half-life 24,110 years) experiments.
plutonium-induced health effects are considered to be the result of energy deposited by alpha particle emissions in tissues that retain plutonium for extended periods (i.e., lung, bone, liver following inhalation exposure). Similar health effects would be expected from any alpha-emitting source that would result in similar cumulative tissue-specific radiation dose and dose rate.
They seem fairly certain that it is alpha particle damage, not chemistry.
The toxicology of soluble heavy-metal bioavailable material is a well understood area of research, and can bioaccumulate in humans several times higher than background if a food chain allows biomagnification of a contaminant. The publication assumes the reader has first year biology, and will understand test subject mortality data implications.
I would recommend studying the story of Polychlorinated biphenyls in whales, Mercury levels in salmon, and PFAS in the Veritasium hosts blood.
https://www.ncbi.nlm.nih.gov/books/NBK599402/