Unlike your first source, this one does not even attempt to quantify what percentage of the variation in ME is explained by genetics. This only says that genetics plays a role. You need strong evidence to credibly state a monocausal explanation for something like ME.
The reality is that different biomarkers were discovered through the years by the Stanford centre by we are still looking for a specific biomarker to ME.
For example one of the criteria to diagnose ME is to have Pots, which is a disease with very specific symptoms.
Pots is a disfunction of the autonomic system and it is often associated with gastroparesis.
I repeat, you are endorsing a very obsolete theory about ME.
This is an unnecessary ad hominem.
> https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3643988
Unlike your first source, this one does not even attempt to quantify what percentage of the variation in ME is explained by genetics. This only says that genetics plays a role. You need strong evidence to credibly state a monocausal explanation for something like ME.