[yarg]

Nah, we've just spent a long fucking time not dying with increasing efficiency.

Distributed autoadversarial algorithms have honed our minds over millions of years.

You may be able to show that something is largely a scaled form, but you're leaving out optimised mass/volume/energy scaling optimisations that have one hell of an impact.

Feedback loops of minor advantages tend to cascade and the propagate across the entirety of breeding populations.

(Some advantages are more aggressively curbed, such as the sickle cell trait.)

Then it's missing things like FOXP2 - weird shit happens to language capabilities when you break it.

Not only that, but:

    Three amino acid substitutions distinguish the human FOXP2 protein from that found in mice, while two amino acid substitutions distinguish the human FOXP2 protein from that found in chimpanzees,[17] but only one of these changes is unique to humans.[10] Evidence from genetically manipulated mice[22] and human neuronal cell models[23] suggests that these changes affect the neural functions of FOXP2.

So there are major mutations that make differences on scales that they aren't even looking at it.

Topological theory has its limits.