[thrusong]

After seeking medical help, my doctor did discover I had a critical B12 deficiency, but AFAIK there is no connection even though I had nerve damage from the B12 issues.

Apparently a healthy number is 500+, most people will present with issues in the 250-300 range, and I was at 166 when critical is 144ish.

I have to take a daily pill for the rest of my life and things have improved, but the VSS is still the same.

[garganzol]

There is a connection. You can imagine a nerve as a cord and when it gets damaged even slightly the signal dropouts do occur. Just like an analog TV gets noisy when the coaxial cord or its connection gets subpar, the human visual field gets distorted in a similar way. You can apply that analogy to hearing problems (e.g. tinnitus) as well.

For a long time we believed that when neurons die the condition stays forever and cannot be improved.

However the consequent observations on WW2 veterans proved the opposite - neuro tissues do recover but they do so extremely slowly.

If you had such a good response to B12 means that not all is lost.

What you can try to achieve is to provide the conditions for faster neurological recovery: speed-up the cellular ATP production to provide the energy for anabolism while slightly suppressing the immune system response so it does not get in the way. You biological machinery will do the rest.

The first part of the equation is reached with specific combinations of vitamins and coenzymes. For example, here is a combination that can serve as the basis: B1 in therapeutical doses (pills, >= 250 mg per day) + multivitamin. The second part of the equation is reached with NSAIDs.

Such treatment may allow you to reach some notable improvements just in few months. And those improvements will persist and accumulate.

[thrusong]

I really appreciate this— I'll look into it!