well I was not, only that between those two stressors, the same major biomarkers are involved. There often is a surprising level of similarities between seemingly distinct things and here I would bet there are possible comparisons however that is not the point of my parent comment, just an answer to which biomarkers are involved.
A more direct comparison would be with the acutes and long terms damages of hypoxia, because oxygen is a similar cofactor bottlenecking energy production, as is the absence of glucose/pyruvate from a fasting human which singlehandely rely of lipid/beta-oxidation.
e.g. https://pubmed.ncbi.nlm.nih.gov/21542052/
But it's not because those are the same mechanism (a bottleneck on a critical cofactor for ATP) that the effects have the same potency because of course fasting is less dangerous in strength than hypoxia.