[oxymoran]

Anecdote: Wife has had very mild covid for 5-6 days now. Been sharing a bong with her and I have no symptoms. I smoke much more than she does. She is boosted. I stopped after the second dose.

Also, I heard somewhere, and I forget where now, that hot smoke could play a role in this, but this study seemed to indicate it was orally bioactive as well. I would assume the in-vivo test wasn’t blowing hot smoke in a test tube but rather bathing in a cannabinoid solution?

[edmundsauto]

As another comment mentioned, this was with non carboxylated cannabinoids. If you’re smoking the leaves then it doesn’t really apply to you.

[somesortofsystm]

Not all cannabinoids in the smoke are carboxylated, or at least lets agree that the efficiency of your average conversion is not 100%. Some degree of non-carboxylated cannabinoids can ride those particles too .. for example at the beginning of a joint, the smoke is being drawn through THC fields which have not yet been burned, but may be dislodged and part of the draw as the heated substrate is pulled into the lungs ..

[edmundsauto]

Agreed, there may be some applicability of this study on smokers (albeit at a reduced effect size). Worth betting on it being protective? Or just a rationalization?

[somesortofsystm]

I don't know if its protective or not, but I do believe its worth finding out.

Wouldn't it be strange if, indeed, weed was the answer to everything.