"In addition, angiotensin converting enzyme 2, the receptor for SARS-CoV-2, is expressed on myocytes and vascular endothelial cells, so there is at least theoretical potential possibility of direct cardiac involvement by the virus"
The most sane hypothesis I've seen is that the virus will attack any cell with the ACE2 receptor, including the heart and kidneys:
"Angiotensin converting enzyme 2 (ACE2) is an enzyme attached to the outer surface (cell membranes) of cells in the lungs, arteries, heart, kidney, and intestines."
This would require the virus to enter the bloodstream from the lungs, which is possible after sufficient alveolar damage.
"In addition, angiotensin converting enzyme 2, the receptor for SARS-CoV-2, is expressed on myocytes and vascular endothelial cells, so there is at least theoretical potential possibility of direct cardiac involvement by the virus"
The most sane hypothesis I've seen is that the virus will attack any cell with the ACE2 receptor, including the heart and kidneys:
"Angiotensin converting enzyme 2 (ACE2) is an enzyme attached to the outer surface (cell membranes) of cells in the lungs, arteries, heart, kidney, and intestines."
This would require the virus to enter the bloodstream from the lungs, which is possible after sufficient alveolar damage.
ref: https://en.wikipedia.org/wiki/Angiotensin-converting_enzyme_...
As an aside, although Wikipedia doesn't mention it, testicular cells also express ACE2, leading to some worry about downstream fertility effects:
https://www.medrxiv.org/content/10.1101/2020.02.12.20022418v...