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As far as I know, 'markers' reflect metabolic/functional attributes in malignant cells. The question about any type of cancer is wether you can target the malignant cells somewhat exclusively or not. Does the cancer retains healthy features as pathways to cell arrest or apoptosis? Is its metabolism so rapid, to make it's growth distinct over a certain time frame? Is its growth influenced by external signalling? Can the immune system find the malignant cells? The problem really is that cancer is often very heterogeneous, which means a targeted treatment might not kill all of it, leading to evolutionary drive and resistant reoccurrence. The more control mechanisms lost, the wilder the mutations. So worst case, you got cells that behave nothing like a normal cells, but generally can't be distinguished/targeted on a cellular level AND are not localized lesions anymore.

So, no, I don't think so. To theoretically transfer a distinct marker to all cancer cells, you'd need a distinct target in the first place.