| What facts? This is not a peer reviewed journal, you know. Here are two distinct facts: 1. I do have eyes and I can see a trend. * I do not recall seeing less fat people as a child, but I do recall seeing more thin people now. * I also notice that the fat people I see now seems on average younger than the fat people I recall from my childhood. * I also notice that the fattest ones - those grossly obese instead of merely overweight - are both larger and more common now than what I recall from childhood (and I was practically raised by an aunt that used to be at the very top of the scale back then, but somehow found herself surpassed in the last 20 years or so). 2. We are told by the media that there are statistics that show an epidemic of overweight. The study pointed out claims that physicians consider overweight as one cause (not the cause) of sleep apnea. We are now told that there is also an epidemic of sleep apnea. * If A causes B, and we observe an increase of both A and B. It is only logic that the "increase of A" is at least partially responsible for the "increase of B". * It would make an interesting scientific study to figure out the degrees of correlation between "increase of A" and "increase of B". It would be even better if such study could conclude if such correlation is strong enough to justify calling "increase of A" a driving factor in the "increase of B" or not. * On the other hand, it is a logical fallacy to claim that no conclusion can be reached regarding the causation/correlation between increases of A and B without "backing it up with facts". I will even claim that it is AntiScientific. If you start with that kind of attitude, you will never be able to form falsifiable hypothesis and therefore will be going through the motions of the scientific method without getting down to the substance. |
The observable results are somewhat consistent with a contagious disease. Adenovirus 36 has been shown to cause long-term obesity in lab animals. Gut microbiome comparative studies have shown a marked difference between the intestinal flora of fat people and skinny people.
Additional environmental factors are in play. Car culture in the US reduces the necessity for daily exercise in comparison to walkable city infrastructure elsewhere in the world. The US uses a lot of chemically altered corn syrup in prepared foods in lieu of other sugars. Agricultural practices differ between the US and other nations, particularly with respect to allowable herbicides and pesticides. Real inflation-adjusted disposable income has been declining in the US since about 1970, and poorer people have less healthy diets. Parenting culture has changed since 1970, such that children left to play outside (aka exercise) without direct adult supervision may actually get a parent arrested. People have increased their consumption of sedentary entertainments, and reduced the time that they typically sleep.
The only way to really figure out which factors contribute most to increases in obesity is by objective analysis of unbiased data. That will lead to actual, testable hypotheses, which will in turn lead to solutions that actually work to reduce the public health risks represented by excess adiposity.
Telling every fat person "hey fatty, go lose some fat" may seem like a simple and easy solution on its face, but it really is a lot more complex and difficult than that.
No one is denying that fat people would be healtier if they were skinnier, or that they do want to be less fat. Very few fat people actually want to be fat, and most do take active measures to try to become less fat. But no one apparently knows with certainty why those measures--often the exact same measures employed by skinny people to avoid becoming fat--do not always work.
When people dismiss those discrepancies as "not trying hard enough" rather than "potential research opportunity", that does not advance the human knowledge in this domain that would allow us to better address obesity on a worldwide scale rather than piecemeal.